分子生物学文献翻译英文

Abstract Objective : Workers peripheral blood lymphocytes of p53 and DNA damage and repair gene mRNA expression levels of benzene exposure and the environment, explore specificity, sensitivity, stability, easy to collect specimens of the advantages of detecting indicators used benzene health care professional Methods : SYBR GreenI chimeric fluorescence quantitative real-time fluorescence RT-PCR analysis, testing workers exposed to 72 (under different posts divided into Group A, Group B) and 29 staff were peripheral blood lymphocytes and related gene p53 mRNA expression Application of differential expression analysis software REST 2005, compared with the control group operating multiple groups to express differences; While peripheral blood leukocytes, hemoglobin and platelet count; Sampling of raw materials toluene, benzene spraying agent content; Workplace air toluene concentration Results : Group A and Group B p53, p21, wild, Ape1 and Mdm2 expression levels compared with the control group was no significant difference, in Group A sword, Bcl-2, Bax, and Xpc Xpa expression level downward, Group B expression edged down compared with the control group was statistically significant A group of white blood cell, hemoglobin and platelet lower than the control group, Group B platelet lower than the control group differences are Conclusion : benzene-exposed workers part of the blood cells with DNA damage and repair gene mRNA expression levels of benzene and non-exposed control group SYBR GreenI chimeric fluorescent real-time RT-PCR and REST 2005 statistical software for detection and analysis of differentially expressed mRNA level, applies to occupational population of molecular epidemiological studies and health

基因工程或者分子生物学类的英文文献翻译来吧。。我这能

VDAC2和醛甲确定膜蛋白的K562 细胞表达增加下铁剥夺 导言 作为一项过渡金属，铁使电子传递和 氧化还原反应。由于这个能力，铁是必要的 所有活的生物体。铁是关键组成部分，许多 细胞蛋白，包括酶和调控蛋白 它所需的细胞过程如DNA 三磷酸腺苷的合成和生产[ 1 ， 2 ] 。 更高层次的蜂窝铁可导致细胞氧化 损伤和死亡。另一方面，缺铁 类似的情况使人缺氧，可诱导细胞凋亡 [ 3 ， 4 ] 。因此，处理铁内的细胞是 严格控制之下。蜂窝铁水平是通过控制 后转录调控的铁运输，储存， 和利用以及描述墨西哥革命制度党（铁规章 蛋白） /爱尔兰（铁反应元件）系统[ 5 ， 6 ] 。安大略 另一方面，铁，本身，似乎是参与转录 调控的几个基因[ 7-10 ] 。此外， 铁可以调节基因表达细胞通过氧化还原状况。 铁的影响转录通过铁产生 活性氧（ ROS ）和铁损耗模拟 缺氧[ 11-15 ] 。结果表明，铁剥夺 可能会导致缺氧诱导因子- 1α激活[ 4 ] 。 铁的能力产生活性氧可能代表 一个重要机制，它可以调节许多细胞 功能，包括铁矿石运输。就是铁铁减少 反应与超氧阴离子自由基，随后有色金属

太佩服了。10分翻译这么多。楼主。多给人几分吧。